By Thomas C. Spoor

There are numerous nice neuro-ophthalmology texts on hand starting from large encyclopedic tomes to small, designated assets. Combining the easiest gains of those books, Atlas of Neuro-ophthalmology bargains a glimpse right into a big range of infrequent and strange neuro-ophthalmic issues. lengthy famous as a number one authority at the optic nerve, Tom Spoor distills his years of expertise into this beautifully illustrated atlas.

The ebook is a totally revised and up-to-date model of Dr. Spoor's unique Atlas of Optic Nerve problems (1992). He addresses all elements of the optic nerve and its disorders-such as optic atrophy, neuropathy, and nerve tumors-making this a necessary e-book for practitioners and scholars. The inclusion of uncomplicated and complex subject matters makes it an awesome examine consultant and the excellent illustrations make it a realistic diagnostic instrument

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Ascending optic atrophy is secondary to a primary retinal disorder destroying ganglion cells. Examples include central retinal artery occlusion (CRAO; Fig. 21), extensive macular lesions (Fig. 21 Optic atrophy due to diffuse loss of retinal ganglion cells after central retinal artery occlusion. 22), infiltration and destruction of ganglion cells (Tay-Sachs and Batten-Mayou diseases; Fig. 23) or diffuse retinal disease (retinitis pigmentosa; Fig. 24). There are a variety of metabolic (mucopolysaccharidoses, lipidoses) and degenerative neurologic diseases (Friedreichs’s ataxia, spinocerebellar degeneration, Charcot-Marie-Tooth disease) associated with optic atrophy and visual loss.

13). Compromise of a short ciliary artery may result in optic nerve head or peripapillary retinal infarction. Excessive damage to the short ciliary arteries may compromise optic nerve function. Obstruction of this blood supply is clinically significant in both arteritic and nonarteritic ischemic optic neuropathy. MYELINATION Myelination of the optic nerve posterior to the lamina cribrosa is accomplished by oligodendrocytes. Myelination insulates an axon, making impulse conduction more efficient.

However, the intracranial optic nerve did appear enlarged and atrophic (Fig. 46d), and was biopsied despite my objections. A primary glioblastoma of the optic pathways was evident on histopathologic examination (Fig. 46e). Always certain; often wrong. Progressive loss of visual acuity or field still demands an explanation. 47 Posterior visual pathways from the lateral geniculate body to the occipital lobe. 48 Lesions of the posterior visual pathways result in bilateral, homonymous visual field defects.

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